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The Reason We Won’t Have Autonomous Cars Any Time Soon

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In the world of autonomous vehicles, Pittsburgh and Silicon Valley are bustling hubs of development and testing. But ask those involved in self-driving vehicles when they might actually be seen carrying passengers in every city, the almost universal answer is: Not anytime soon.
No current regular-production vehicle should be driven hands free (though there is plenty of footage on YouTube of Tesla drivers doing just that). Part of the reason is that the current semi-autonomous vehicles receive no information from either infrastructure (traffic lights, railroad gates, etc.) or surrounding vehicles.
Even then, there would be older vehicles not yet retired from service gumming up the works. What impact these cars and trucks would have on an otherwise autonomous-ready fleet is difficult to say, but we can assume that even 15 years from now, pure autonomy will likely be a limited condition.
As of this writing, only 10 states have adopted legislation related to the operation of autonomous vehicles. And in each case, those laws relate to the testing of said vehicles on public roads, not the sale and consumer use of them.
The truth is, no matter how autonomous driving becomes, vehicles will still need to be equipped with a steering wheel, throttle and brake pedals, and full instrumentation. This is because human drivers will still be called upon from time to time to take the controls.
With all of the developments in self-driving cars by companies such as Tesla, Waymo, and others you would think that autonomous vehicles would be on the road by now. However, such vehicles are not becoming mainstream and people are wondering what some of the reasons are. Therefore, we would like to explain what is holding up the adoption of self-driving cars and are their solutions to these issues.
Self-driving cars are equipped with AI-powered cameras to help them navigate through the streets and identify some of the objects, road signs and pedestrians they encounter on the road. However, after a heavy snowstorm the camera cannot see the delineation markings on the roads thus making it almost impossible to drive safely. As of today, researchers have not found a way to solve this problem, which is why a lot of the testing is done in warm-weather climates such as San Diego and Los Angeles.
We can add to this the general antagonism that human drivers have towards self-driving cars. In fact, police in one city in Arizona documented at least 21 cases of people harassing Waymo vehicles with one person actually pointing a gun at it.
We mentioned a little bit earlier the general antagonism that people have towards self-driving cars, but the problem exacerbated when we talk about fatalities. A couple of years ago, there was an Uber crash near Phoenix, AZ involving a self-driving car. A poll by AAA determined that 73% of drivers were reluctant to give up control of their vehicles.
We hope that the information presented gave you more insights about why self-driving vehicles have not gone mainstream. Mindy Support is dedicated towards providing researchers with data annotation assistance to help them overcome the problems mentioned above. As AI advances and new developments are made, you can expect to see self-driving cars on the road sooner than most people think.
The idea is that Tesla keeps improving the system by testing through its FSD beta fleet and collecting data to train its neural nets. With enough improvements, Tesla hopes that its FSD system will eventually become safe enough to use without drivers monitoring it, but there have been doubts about making it happen after several missed timelines.
Finally, several smaller companies, including Zoox, a robo-taxi company; Ike, an autonomous-trucking startup; and Voyage, a self-driving startup; have also passed the torch to companies

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In flowering plants, floral longevity is species-specific and is closely linked to reproductive strategy; petal senescence, a type of programmed cell death (PCD), is a highly regulated developmental process. However, little is known about regulatory pathways for cell death in petal senescence, which is developmentally controlled in an age-dependent manner. Here, we show that a NAC transcription factor, designated EPHEMERAL1 (EPH1), positively regulates PCD during petal senescence in the ephemeral flowers of Japanese morning glory (Ipomoea nil). EPH1 expression is induced independently of ethylene signaling, and suppression of EPH1 resulted in Japanese morning glory flowers that are in bloom until the second day. The suppressed expression of EPH1 delays progression of PCD, possibly through suppression of the expression of PCD-related genes, including genes for plant caspase and autophagy in the petals. Our data further suggest that EPH1 is involved in the regulation of ethylene-accelerated petal senescence. In this study, we identified a key regulator of PCD in petal senescence, which will facilitate further elucidation of the regulatory network of petal senescence. © 2014 The Authors The Plant Journal © 2014 John Wiley & Sons Ltd.
Senescence violates the most basic tenet of natural selection by causing death rather than individual survival. Thus, current theories favor the concept of antagonistic pleiotropy (AP) to explain how aging emerged in metazoans. Presumably, pleiotropic genes reduce vigor and limit longevity in adults. However, they also promote fitness and reproduction in juveniles, causing them to be selected and retained in the gene pool. The general hypothesis presented herein is a special case of AP that identifies the common cause and mechanism of aging in iteroparous (i.e., capable of reproducing multiple times) animals. It ascribes senescence to unremitting, nonprogrammed change or remodeling forced upon the adult soma by postmaturation expression of developmental gene(s) affecting dynamic transformation of the single-celled conceptus into a complex, multicellular organism. Whereas persistent somatic change is necessary for development to proceed normally, it also has the potential to erode homeostasis in adults after maturation is complete. Thus, developmental inertia is the primary cause of senescence, whereas decay of internal order and integrated function among interdependent systems of the body is the general mechanism by which aging progresses over time. Accordingly, this global pathogenic process creates an environment in which the many recognized, age-associated physiologic and metabolic sequelae can arise as consequences of senescence rather than causes of it. Paradoxically, the genes that promote somatic remodeling essential for development and survival also guarantee aging and death by the same action whose outcomes differ only by the time it is expressed relevant to maturation.
Senescence represents the final stage of leaf development but is often induced prematurely following exposure to biotic and abiotic stresses. Leaf senescence is manifested by color change from green to yellow (due to chlorophyll degradation) or to red (due to de novo synthesis of anthocyanins coupled with chlorophyll degradation) and frequently culminates in programmed death of leaves. However, the breakdown of chlorophyll and macromolecules such as proteins and RNAs that occurs during leaf senescence does not necessarily represent a one-way road to death but rather a reversible process whereby senescing leaves can, under certain conditions, re-green and regain their photosynthetic capacity. This phenomenon essentially distinguishes senescence from programmed cell death, leading researchers to hypothesize that changes occurring during senescence might rep

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