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The adipose tissues (ATs) are endocrine and dynamic organs that display high morphological and functional plasticity. White AT (WAT) was named that way because it presents white adipocytes in its composition. In contrast, brown AT (BAT) has as its main integrant the brown adipocytes [1]. Both ATs play various physiological roles, including energy storage, endocrine regulation, and thermogenesis. As a means of adapting, mammalians developed a mechanism to maintain their body temperatures under unfavorable climates [2]. This process, called adaptative thermogenesis, occurs due to the elevated plasticity of ATs, which allows reversible changes in their morphology and functions [3]. Studies have shown the capacity of progenitor cells as well as mature adipocytes to differentiate into a model that presents similarities with the brown profile, called brite or beige AT [4,5,6]. When this phenomenon occurs in mature adipocytes it is called browning of WAT [4].
The increase in energy expenditure can act as a therapeutic approach to metabolic syndrome, and also can be associated with poor prognosis of diseases associated with hypermetabolism [15,16,17,18]. For this reason, efforts have been employed to identify the key participants in the regulation of browning. This review aims to present and describe the current studies related to both endogenous and exogenous most relevant agents and their biological mechanisms at biochemical and molecular levels.
On the other hand, the transdifferentiation hypothesis proposes beige cells arise from mature white adipocytes, in a reversible process, after adipogenic stimulus without the participation of a progenitor-like state of cells [37]. The underlying molecular mechanisms for transdifferentiation are under intensive research, but some studies already show that this plasticity process occurs mainly in scWAT depots [29]. Known as browning, this process has gained increasing attention in the research area as an alternative method of energy stimulation. UCP1 expression can be stimulated when white adipocytes are exposed to stimuli, previously referred as to adipogenic stimulus, [20, 27, 29], driven by a set of molecules known as browning markers.
In contrast, the upregulation of UCP1 or even only its activation can perform a paradoxical role in hypermetabolic scenarios and associate with a worse prognosis [76, 77]. It is proven that diet-induced whitening is related to the upregulation of this gene. A greater expression of browning markers (e.g., UCP1, PGC-1α, TBX1) was found in obese human patients mainly in vWAT [78] Besides that, mice affected by cancer-associated cachexia tend to show increased thermogenesis gene expression and BAT activation [79].
The browning process is spontaneously induced by tumor-secreted factors and IL-6 during cachexia development, which can lead to full depletion of AT [80, 81]. Interestingly, the elicitation of browning after burn injury is associated with the hypermetabolic response, as well as an increase in lipolysis and free fatty acid efflux that can outcome in liver steatosis [82, 83]. In addition to the therapeutic impact of the browning process in obesity and metabolic diseases, recent discoveries regarding the impact of UCP1-dependent BAT activity in hypermetabolism conditions should be further investigated in the context of UCP1 to appropriately regulate browning for application in different situations.
Initial studies demonstrated that WAT UCP1 mRNA and protein levels are strongly decreased in β3-AR knockout (KO) mice [30, 93]. In addition, β3-AR agonists are well-known for inducing ectopic UCP1 expression in WAT coupled with a significant mitochondrial enhancement in rodents, and for augmenting glucose homeostatic activity of their BAT [84, 94]. On the other hand, in humans, early efforts to increase browning activation with the use of β

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id: 7000878fps: 23.976nb_cd: 1format: srtchar_encoding: feature_type: Movierelease_name: Burn.After.Reading.2008.1080p.BluRay.x264.DTS-FGTrelease_title: release_group: video_codec: screen_size: video_format: author_comments: Based on 8163291. Changed the framerate and retimed (add 500msec delay).bad: 0enabled: truevotes: 0points: 0ratings: 0.0thanks: 7download_count: 291last_download: 2023-02-05 06:41:04 UTCupload_date: 2023-01-23 09:51:07 UTCcomments_count: 0favourited: 0auto_assign: falsehearing_impaired: falselast_comment: featured: falsefeatured_ts: hd: trueauto_translation: falseforeign_parts_only: falsefrom_trusted: falseadmin_check: falseadmin_checked: trueversion: 1translator: detected_language: Japanesedetected_encoding: UTF-8smart_feature_name: 2008 - Burn After Readingosdb_subtitle_id: 9404445subfiles_count: 1osdb_user_id: 9637597osdb_user_info: "display_rank"=>"Application Developers", "display_user"=>"os-auto", "display_author"=>"Anonymous", "uploader_badge"=>"platinum-member", "display_user_email"=>"no-email-user-cof7cjps@email-ls1t.com", "display_author_rank"=>"anonymous"slug: 9404445-burn-after-reading-2008-1080p-bluray-x264-dts-fgtuploader_id: 3282translator_id: created_at: 2023-01-23 09:06:08 UTCupdated_at: 2023-02-05 11:10:48 UTCfeature_id: 541937osdb_language_id: 1152ai_translated: falseparent_subtitle_id: childrens_count: 0new_download_count: 9new_last_download: 2023-02-05 00:00:00 UTClast_sync_files: 2023-02-05 11:10:39 UTC
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Determines if the server should burn in subtitles when converting video depending on the subtitles format. Avoiding burning in subtitles improves server performance and is only supported by the HTML5 player. Available choices:
Note that the subtitles video filter opens the file separately, so if you are also Seeking in the same step, then the subtitles will start at the beginning of the subtitle file. This may or may not be what you want. If you want the burned-in subtitles to start at the same timestamp as the video, you will need to copy the timestamps to the output with -copyts, and then additionally seek in output again with the same values that were used for the input. For example, if you wish to start both the output video and subtitles to start 5 minutes into the input file, then you can use a command line this:
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In this fairy tale with a sexy twist, she’s a penniless San Francisco seamstress. He’s the king of Italian couture. Who’s got designs on whom?Boutique owner Kimber DiSanto has seen better days. She’s been dumped at the altar by Prince Charmless, her business went up in flames (literally), and now she’s stuck in Florence, Italy, with an ice-queen stepmother, to try to save her late father’s failing dress shop. Only one thing could make it worse: another man in her life. The arrogant Italian fashion tycoon offering to buy her father’s shop is as rich as he is sexy, and their attraction is off the charts. But Kimber’s not about to get burned again.Women don’t say no to Matteo Moretti—and certainly not with Kimber’s stinging precision. With all the heat and fury sparking between them, Matteo can’t resist baiting the gorgeous American. His plan? Win her over one scorching kiss at a time.Kimber tells herself it’s all just a game. That her broken heart isn’t in danger, and that Matteo’s touch does not make her Lady Land dance with joy. But sometimes it takes the fieriest of enemies to turn a fantasy into a real-life romance."
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